thyroid confusion

[Jean]

I’ve got primary hypothyroidism since 20 year’s. This first 12 year’s of levothyrox is feel good. After this time levothyrox don’t work very well, because my adrenal shut down.

I don’t known why my adrenal shut down but I’ve a suspicion about pathogens (lyme disease, candidiasis…)

Today, I take armour + Levothyrox + HC and some pregnenolone. But, I’ve difficulty to lose weight and a lack of energy depiste a good diet and exercise program.

Thank you for the Dr Marianco answer because my ferritin is to low…. I try to optimize the level

But I have a suspicion about the best thyroid treatment. In fact, if I take only armour I feel bad if I take 2 grains, I lose muscle size and no fat, and more tired depiste hydrocortisone therapy

If I take a mix of one grain of armour with 150 mcg of T4 (levothyrox) my blood level is high normal range but I don’t lose weight depiste normal temperature

Free T3 —- 3, 98 pg/nl (N : 2,3 to 4,2)
Free T4

---

24,14 ng/l (N: 8,90 to 17,6)
TSH 0,010
RT3

---

0, 25 ng/ml (N: 0,09 to 0,35)

In fact, thera a war between doctor that explain armour work best because it’s natural and some doctor that explain that armour is to bad because is increase to much free T3

There are some study that explain that levothyrox is toxic for the liver (decrease glutathion level)

Thank you for your advises

Endocr J. 1999 Aug;46(4):579-83. LinksLevothyroxine-induced liver dysfunction in a primary hypothyroid patient.
Ohmori M, Harada K, Tsuruoka S, Sugimoto K, Kobayashi E, Fujimura A.
Department of Clinical Pharmacology, Jichi Medical School, Tochigi, Japan.
Here we report a case of levothyroxine-induced liver dysfunction. T4 (levothyroxine) has been more commonly used for the treatment of hypothyroidism than T3 active hormone (triiodothyronine), because with the former drug a stabler plasma concentration is obtained after oral administration. Although there are few reports on levothyroxine-induced liver dysfunction, we treated a primary hypothyroid patient with high serum aminotransferase after administration of levothyroxine. Liver dysfunction was improved after cessation of the drug administration. Antibody to T4 was found in the serum of the patient after this event. From clinical course and laboratory data of the patient, the episode of liver damage was considered to be induced by levothyroxine. We then administrated triiodothyronine, and it did not induce liver dysfunction. Changing levothyroxine to triiodothyronine resulted in a successful clinical course in this case, as re-administration of the doubtful drug is strictly limited.
Intern Med. 2007;46(14):1105-8. Epub 2007 Jul 17. Links
Liver injury induced by levothyroxine in a patient with primary hypothyroidism.
Kawakami T, Tanaka A, Negoro S, Morisawa Y, Mikami M, Hojo M, Yamamoto T, Uegaki S, Aiso M, Kawasaki T, Ishii T, Kuyama Y, Fukusato T, Takikawa H.
Department of Medicine, Teikyo University School of Medicine, Tokyo.
We report a patient with primary hypothyroidism, who developed hepatocellular injury due to levothyroxine, synthetic thyroxine. A 63-year-old male was admitted to our hospital due to elevation of liver enzymes. The patient was diagnosed as having hypothyroidism and had been treated with levothyroxine for almost two months until admission. Drug-induced liver injury induced due to levothyroxine was suspected and liver enzymes were rapidly decreased after discontinuation of levothyroxine and dried thyroid powder, also containing thyroxine. Synthetic triiodothyronine, the deiodinated form of levothyroxine was administered instead, and was well tolerated by the patient. The drug-induced lymphocyte stimulation test (DLST) using levothyroxine was negative. Since triiodothyronine which structurally resembles levothyroxine did not cause liver injury, and DLST using levothyroxine was negative, it is unlikely that levothyroxine itself was targeted by the immune system. Rather, we assume that the complex of levothyroxine as the hapten and liver-related macromolecules in the body as the carrier might have acquired antigenicity in this patient and subsequently resulted in liver injury

[DrMariano2]

@Jean 355 wrote:

I’ve got primary hypothyroidism since 20 year’s. This first 12 year’s of levothyrox is feel good. After this time levothyrox don’t work very well, because my adrenal shut down.

I don’t known why my adrenal shut down but I’ve a suspicion about pathogens (lyme disease, candidiasis…)

Today, I take armour + Levothyrox + HC and some pregnenolone. But, I’ve difficulty to lose weight and a lack of energy depiste a good diet and exercise program.

But I have a suspicion about the best thyroid treatment. In fact, if I take only armour I feel bad if I take 2 grains, I lose muscle size and no fat, and more tired depiste hydrocortisone therapy

If I take a mix of one grain of armour with 150 mcg of T4 (levothyrox) my blood level is high normal range but I don’t lose weight depiste normal temperature

Free T3 —- 3, 98 pg/nl (N : 2,3 to 4,2)
Free T4

---

24,14 ng/l (N: 8,90 to 17,6)
TSH 0,010
RT3

---

0, 25 ng/ml (N: 0,09 to 0,35)

In fact, thera a war between doctor that explain armour work best because it’s natural and some doctor that explain that armour is to bad because is increase to much free T3

There are some study that explain that levothyrox is toxic for the liver (decrease glutathion level)

Thank you for your advises

Endocr J. 1999 Aug;46(4):579-83. LinksLevothyroxine-induced liver dysfunction in a primary hypothyroid patient.
Ohmori M, Harada K, Tsuruoka S, Sugimoto K, Kobayashi E, Fujimura A.
Department of Clinical Pharmacology, Jichi Medical School, Tochigi, Japan.
Here we report a case of levothyroxine-induced liver dysfunction. T4 (levothyroxine) has been more commonly used for the treatment of hypothyroidism than T3 active hormone (triiodothyronine), because with the former drug a stabler plasma concentration is obtained after oral administration. Although there are few reports on levothyroxine-induced liver dysfunction, we treated a primary hypothyroid patient with high serum aminotransferase after administration of levothyroxine. Liver dysfunction was improved after cessation of the drug administration. Antibody to T4 was found in the serum of the patient after this event. From clinical course and laboratory data of the patient, the episode of liver damage was considered to be induced by levothyroxine. We then administrated triiodothyronine, and it did not induce liver dysfunction. Changing levothyroxine to triiodothyronine resulted in a successful clinical course in this case, as re-administration of the doubtful drug is strictly limited.
Intern Med. 2007;46(14):1105-8. Epub 2007 Jul 17. Links
Liver injury induced by levothyroxine in a patient with primary hypothyroidism.
Kawakami T, Tanaka A, Negoro S, Morisawa Y, Mikami M, Hojo M, Yamamoto T, Uegaki S, Aiso M, Kawasaki T, Ishii T, Kuyama Y, Fukusato T, Takikawa H.
Department of Medicine, Teikyo University School of Medicine, Tokyo.
We report a patient with primary hypothyroidism, who developed hepatocellular injury due to levothyroxine, synthetic thyroxine. A 63-year-old male was admitted to our hospital due to elevation of liver enzymes. The patient was diagnosed as having hypothyroidism and had been treated with levothyroxine for almost two months until admission. Drug-induced liver injury induced due to levothyroxine was suspected and liver enzymes were rapidly decreased after discontinuation of levothyroxine and dried thyroid powder, also containing thyroxine. Synthetic triiodothyronine, the deiodinated form of levothyroxine was administered instead, and was well tolerated by the patient. The drug-induced lymphocyte stimulation test (DLST) using levothyroxine was negative. Since triiodothyronine which structurally resembles levothyroxine did not cause liver injury, and DLST using levothyroxine was negative, it is unlikely that levothyroxine itself was targeted by the immune system. Rather, we assume that the complex of levothyroxine as the hapten and liver-related macromolecules in the body as the carrier might have acquired antigenicity in this patient and subsequently resulted in liver injury

Interesting case, but this is a rare case of someone developing an immune reaction to T4 (Levothyroxine).

Levothyroxine (T4) is identical to what the thyroid gland makes. Synthetic or naturally made, it is the same substance. To myself, Levothyroxine is a bioidentical hormone treatment because it is identical to what the body makes.

Armour thyroid is 20% T3 (triiodothyronine) and 80% T4 (levothyroxine). Thus if one is taking Armour Thyroid, then one is also taking Levothyroxine.

The problem of Armour Thyroid alone treatment is that it has a higher T3 component than what the human thyroid gland produces. Thus, some people may be more sensitive to the higher T3 component and won’t do well. On the other hand, many people do better on Armour Thyroid because they need the higher T3 component than what could be obtained with Levothyroxine treatment alone. And some people do better on a combination of Armour Thyroid and Levothyroxine. This combination reduces the T3 component of treatment. Finally, some people do best on T3 treatment alone – neither Levothyroxine or Armour Thyroid.

The ultimate question is: What mix of T4 and T3 would serve the patient best? That will depend on that individual.

This is difficult to measure since the brain and body are two separate compartments. The brain can have a different conversion rate of T4 to T3 than the body. Thus, the body may have enough thyroid but the brain can be hypothyroid, causing significant problems in function. Measuring brain levels of thyroid hormone is not usually done. It can only be obtained by getting a lumbar puncture from a neurologist then measurement of CSF thyroid levels would be done. This has its own risks.

[Jean]

Thank you for your answer but MANY people and doctors prefer to give armour, there are a web site that explain for hypothyroid people why T4 don’t work
I put in red the recommendation of this post
Myself I do best if I mix levothyrox and armour

http://www.stopthethyroidmadness.com

These are the most common mistakes patients, or their doctors, make when a switch has been made to Armour/Naturethroid/Thyroid-S, etc. and YOU can avoid them by being familiar with these reasons and making sure your doctor understands them, too. Can you find yourself below?
STICKING WITH TOO LOW A DOSE. For a myriad of reasons, this happens often. Have one of these been true of you?
1) being held on a starting dose (such as one grain, less, or slightly more) longer than two weeks
2) being bound by the directives of a TSH-obsessed doctor
3) failing to get a raise of desiccated thyroid until the “next labwork”, which can be weeks and months away
4) following an inaccurate Synthroid-to-Armour conversion equivalence chart
5) being forced to lower a dose due to a high free T3 with continuing hypo symptoms, which is a sign of low cortisol, not too much desiccated thyroid, or
6) being afraid to go higher!

For example, a patient makes her way up to 1-2 grains, notices great improvements, but also has continuing problems. OR, a patient makes her way up to 2 grains and notices NO improvement. And it’s common to think that desiccated thyroid is not working! In reality, it may simply mean a patient isn’t on enough! By observation, many patients seem to need 3-5 grains before completely ridding themselves of symptoms, though some are lower and some higher. It can also be very wise to check adrenal function, since low cortisol can prevent thyroid hormones from making it to the cells….and you will still feel bad.

BEING ON AN OPTIMAL DOSE and FEELING GREAT, BUT BEING LOWERED DUE TO THE TSH LAB RANGE Similar to #2 above, this is the person that made his/her way up to an optimal dose, or the dose that simply made them feel very good and removed symptoms, but having the dose lowered by a doctor who saw your suppressed TSH (i.e. below the range). This is doctor who thinks that ink spots on a piece of paper tell the truth more than your symptoms! When on an optimal dose of desiccated thyroid (or being very near), you WILL have a suppressed TSH without being hyper. Being lowered is a WRONG move.
THINKING YOU ARE ON TOO MUCH BECAUSE OF HYPER-SYMPTOMS Yes, a doctor can guide you to go too high with desiccated thyroid and you’ll have hyper symptoms. You would then want to decrease your amount. But even more common is having hyper-like symptoms (anxiety, shakiness, fast heart rate, etc), especially on doses lower than 3 grains, because of underlying low-functioning adrenals (i.e. not enough cortisol), or even a low Ferritin—-each and/or both of which can be quite common in hypothyroid patients. So it can be wise to get both your Ferritin and cortisol levels checked. Ferritin is easily checked via a blood test, and if a patient is below 50, it can cause problems. As far as Cortisol levels, patients have found that the most accurate test is NOT a one-time blood test, but a 24 hour adrenal saliva test, which catches your levels during a 24 hour period. If money is an issue, try Discovery Steps One and Two on the Adrenal Info page on this site.Additionally, if you raise too quickly, or in too large a dose raise, your body can overreact, making you think you were on too much. The solution for patients has been to go back where they were, and go up in smaller increments, such as 1/4 grain.
FAILING TO MULTI-DOSE Occasionally, some patients take their natural thyroid all at once in the morning and say they do fine. But… most individuals will notice much better results by multi-dosing. For example, a person on 3 1/2 grains might take 2 grains in the morning, one grain by noon or in the early afternoon, and 1/2 grain by mid-afternoon. Multi-dosing better imitates what your own thyroid would be doing, and gives you the direct T3 throughout the day when you most need it. Spreading out the Armour also prevents stress on your adrenals.
SWALLOWING NATURAL THYROID WITH ESTROGEN, CALCIUM or IRON. Estrogen, calcium and iron bind some of the thyroid hormones and makes them unusable. So… it’s wise to avoid swallowing these at the same time you swallow your natural thyroid.
STAYING ON A STARTING DOSE TOO LONG. The key to understanding this mistake is with the word “starting dose”. When first starting on any natural desiccated thyroid product, it can be wise to start on one grain or less, which is lower than you will ultimately need. Why? To help your body adjust to the direct T3. BUT… patients have found it UNWISE to stay on that low dose much longer than 2 weeks without raising. Why? Because hypothyroid symptoms can return with a VENGEANCE due to the feedback loop between the hypothalamus, pituitary and thyroid gland.
THINKING DESICCATED THYROID IS NOT WORKING WHEN SOMETHING ARISES. Desiccated thyroid contains direct T3, and the T3 can initially aggravate certain conditions. When this happens, doctors hae had patients stop the increase of their desiccated thyroid, or decreased it to give the reaction time to go away. An example is Mitral Valve Prolapse–one patient noted that with each raise, she had palps. But they went away within the first 5 days after each raise. One gal got itchy when she got on Armour, and was so determined to blame Armour that she got off, got back on Synthroid, and is STILL itchy.
ADDING T4 or T3 to DESICCATED THYROID, OR EVEN TOO SOON! Most patients report that they do perfectly fine on desiccated thyroid alone, especially when they have taken the time to raise and find their optimal dose, which is often over 3 grains and has removed all hypothyroid symptoms. But some patients and their doctors feel the need to add either synthetic T4 or T3 to their natural thyroid dose to achieve a certain result . The challenge is in not adding it too soon, otherwise you miss out on the benefits of the T4, T3, T2, T1 and Calcitonin. Instead, if they had simply upped their desiccated thyroid more, they might have gotten the results they desired. Occasionally, a patient may suspect they have thyroid hormone resistance when 5-6 grains of desiccated thyroid is not doing the job. At this point, they add T3, or Cytomel, to their dose, to achieve results. Addtionally, since most patients on an optimal dose of desiccated thyroid only achieve a mid-range T4, some are adding a small amount of T4 to raise the level.
GOING UP WITH DOSAGES WAY TOO FAST. This was observed a few years ago: a patient got on Armour. One grain, then 2 grains, 3 grains, 4 grains, 5 grains, then 6 grains. But the problem was that he did this within 4-5 weeks! OUCH. He started to find himself majorly overdosed with symptoms to match (high heart rate, sweating). He had to stop for a few weeks… then resume again at one grain and do it the right way. Namely, patients have noted that after they have been on a starting dose for a few weeks, they can start rising by 1/2 grain or so every 2-3 weeks. It’s all individual, and some may need lower amounts, but that seems to be the general amount to raise. They also note that when they get up to 2-3 grains, it’s time to hold each dose at least 4-6 weeks to allow the buildup of the T4, and to see it’s conversion to T3 results.
PAYING TOO STRICT ATTENTION TO LABS. As mentioned above, thyroid patients have noticed that doctors tend to treat lab results rather than treat PATIENTS. Labs are interesting, and labs are good adjuncts to the full spectrum of dosing. BUT… SYMPTOMS are IMPORTANT. For example, patients have learned that even if there is a very suppressed TSH, and/or a high free T3, yet symptoms continue, it’s important to look at one’s adrenal function, since low cortisol can make the free T3 go high while symptoms continue.
BELIEVING THAT DESICCATED THYROID IS “HARD TO REGULATE”. Totally and completely false. Nothing is hard about desiccated thyroid. You simply raise it high enough to rid yourself of symptoms, which in turn gives you a free T3 towards the top of the range and a suppressed TSH. Believing that desiccated thyroid is hard to regulate is akin to believing that tricycles are hard to ride.
THINKING THAT SYNTHETIC T4 ALONG WITH SYNTHETIC T3 (aka Cytomel), OR THE COMBO OF THE TWO (Thyrolar) IS JUST AS ADEQUATE AS NATURAL DESICCATED THYROID Adding synthetic T3 to your Synthroid, Levoyxl or other T4 brands is definitely a step up from being on T4 alone! We applaud that addition. But….to say it’s equal to being on desiccated thyroid t’ain’t so. Too many patients who have been on the synthetic combo, and switched to desiccated thyroid, report that the results were even better. That’s impressive. Besides, with desiccated thyroid, you are getting exactly what your own thyroid gives you–T4, T3, T2, T1 and calcitonin. Makes a difference.
THINKING YOUR DOCTOR KNOWS MORE THAN YOU DO. Granted, we have great respect for education, and we appreciate the knowledge that a medical school trained doctor brings to our health quest. It’s important! BUT… that education does NOT take away from our OWN knowledge and our OWN intuitive sense about our bodies… about what works, about what doesn’t work… no matter what that doctor says. This website, and even more the STTM book, represents just that! So, patients have discovered that the doctor-patient relationship is best as a TEAM, with respect going BOTH directions. Doctors are not “gods”. They can and DO make mistakes in judgment. TEAMWORK counts. Find a good doc!!
Want to order your own labwork?? STTM has created the right ones just for you to discuss with your doctor. Go here: https://sttm.mymedlab.com/

Need help interpreting your lab results? Go here: http://www.stopthethyroidmadness.com/lab-values/

[DrMariano2]

I keep an open mind. I am pretty flexible.

I use Armour Thyroid, Levothyroxine, and Cytomel (T3) with my patients.

I am not fixed on using Armour Thyroid alone. Armour Thyroid is also not tolerable to some patients. It goes both ways.

I am not wedded to TSH. TSH is often wrong. Too often wrong. I primarily gauge thyroid function by direct thyroid hormone levels (e.g. Total T4, Free T3) and by the history and physical exam – such as for signs and symptoms of hypothyroidism.

True, Levothyroxine may not work for many people. I usually find that this is because of the presence of a non-thyroid illness. This includes mental illness or brain illnesses, where T4 to T3 conversion is impaired, though body conversion is O.K. (such as Alzheimer’s Disease). Non-thyroid illness includes thyroid resistance syndromes, adrenal problems, nutritional deficiencies, etc.

In fact, many of my patient cannot get Armour Thyroid or Cytomel (T3) at all even if it is indicated. Their health insurance won’t pay for it. They can’t afford it. Their pharmacy cannot get Armour Thyroid or its generics at all since their distributer doesn’t carry it.

With these patients, I have been forced to use Levothyroxine since it is the only available thyroid hormone treatment.

I found, however, that I can get Levothyroxine to work pretty well by addressing the non-thyroid illness itself for these patients.

The presence of the non-thyroid illness that affect thyroid function is what the other alternative thyroid sites, such as stopthethyroidmadness.com, often refer to as a reason that Levothyroxine does not work.

The problem I have is that by treating the non-thyroid illness itself, I am finding that I can get Levothyroxine to work.

In fact, once I treat the non-thyroid illness, I often have to LOWER the dose of Levothyroxine because the patient becomes hyperthyroid if I don’t.

I use to use high dose thyroid in many patients – be it Armour Thyroid or Levothyroxine. High dose thyroid hormone is a textbook psychiatric treatment for mood disorders such as bipolar disorder. However, when I address the rest of the system, when I treat the non-thyroid illness, I am finding that high dose thyroid becomes an incorrect treatment. I am forced to reduce the dose since the patient becomes hyperthyroid or develops hypothalamic-pituitary-adrenal axis dysregulation. This would then destabilize the illness I am trying to treat.

As I get the rest of the system in more optimal signaling, metabolic, and nutritional state, I am finding Levothyroxine at 100-150 mcg a day sufficient to optimize thyroid function in many if not most of the patients I treat with Levothyroxine alone. This is analogous to my use of growth hormone in the treatment of adult growth hormone deficiency. When I optimize the rest of the system, I find I need very low doses of growth hormone to improve function (which as a side effect greatly reduces the cost of growth hormone treatment).

This result completely unexpected and actually shocked the hell out of me. But it makes sense. When non-thyroid illness is addressed completely, one can use standard thyroid treatment that endocrinologists almost universally use – or no thyroid hormone at all if initial thyroid hormone levels are sufficient because thyroid hormone works again.

The more experience I have in treating non-thyroid illness, the less often I end up using high dose thyroid treatment.

Realize that I am a psychiatrist. Every mental illness is a possible non-thyroid illness that can affect thyroid function. Anorexia nervosa, for example, is a good example where Free T3 is low and Total T4 is low and TSH can be low. Treating the mental illness itself can normalize thyroid function or reduce the need for thyroid treatment.

I am not disparaging sites such as stopthethyroidmadness.com. I think they do a service for patients that have found no improvement with conventional treatment using Levothyroxine. I love using Armour Thyroid as a treatment.

The situation, however, I found myself in, of being forced to use Levothyroxine alone with my patients with hypothyroidism forced me to develop assessments and treatments of the non-thyroid illness they have that affected thyroid function. Once I did that, I found I could use Levothyroxine in conventional doses or even not use thyroid hormone in treatment at all if they have sufficient initial thyroid hormone levels.

This is where I figured out that if I kept T4 over 8.0 and addressed the non-thyroid illness, the patient could often return to a euthyroid state, because addressing the non-thyroid illness could get thyroid hormone to work again.

If I can’t address the non-thyroid illness because not every non-thyroid illness has a treatment or is even known, I would then use thyroid hormone at the dose that works – be it Armour Thyroid, Levothyroxine, T3 or whatever combination is needed.

[Jean]

thank you for your answer.

I don’t feel great with only armour, stop the thyroid madness explain that many people need 3 grains, but with only two grains I feel bad (exhausted, water bloat (from increase estrogen), muscle loss. My blood test on armour only is VERY high T3 with low T4.

My brain prefer high T4 level. In fact, when I read some study of Bauer for bipolar disorder, the patients feel better with high dose of levothyrox. The depression is clear after 250 mcg of synthetic T4, bauer said that the brain prefer T4 to make T3.

[chaos]

Doc, do you think there is a benefit from Armour resulting from the T2, T1 and calcitonin it contains?

[DrMariano2]

@Jean 537 wrote:

thank you for your answer.

I don’t feel great with only armour, stop the thyroid madness explain that many people need 3 grains, but with only two grains I feel bad (exhausted, water bloat (from increase estrogen), muscle loss. My blood test on armour only is VERY high T3 with low T4.

My brain prefer high T4 level. In fact, when I read some study of Bauer for bipolar disorder, the patients feel better with high dose of levothyrox. The depression is clear after 250 mcg of synthetic T4, bauer said that the brain prefer T4 to make T3.

In treating depression, T3 has more evidence for effectiveness than T4. Perhaps in many cases brain T4 to T3 conversion is impaired, compared to body T4 to T3 conversion, accounting for the improved effectiveness of T3 versus T4.

In bipolar disorder, T4 at high doses helps stabilize mood. That is a textbook treatment. Of course, from my experience, nearly every person with bipolar disorder exhibits signs of hypothyroidism. Such patients also would tend to have low free T3. The TSH would range from “hyperthyroid” to euthyroid to hypothyroid. TSH is thus not a good measure. From my point of view, the hypothyroidism, itself, is either due to thyroid illness or non-thyroid illness. But either way, it is one signaling problem that is part of the pathophysiology of bipolar disorder.

Returning to the point of this thread:

It is important to maintain flexibility in what a patient may need. Unfortunately, this may need trial and error. Some patients may need T3, T4 (Levothyroxine), Armour Thyroid or a combination of two or three of these to feel their best.

It would be interesting to know what your total T4 is when you feel your best on Armour Thyroid and Levothyroxine.

[Jean]

it’s not clea, but when the winter I increase the dosage to 200 mcg with one grain of armour, I feel good
the wheather when it’s too hot I cut to 125 mcg with one grain, I don’t feel so good. May be when i’s to hot the conversion of T4 to T3 is low !!!

[DrMariano2]

@chaos 539 wrote:

Doc, do you think there is a benefit from Armour resulting from the T2, T1 and calcitonin it contains?

Calcitonin may be useful for osteoporosis. It may have a slight effect on reducing appetite.

T2 and T1 have very mild effects compared to T3.

The effects of these components are fairly mild overall.

[Jean]

What the most stronger T3 ? T3 from armour or synthetic T3, or is the same ?

[DrMariano2]

@Jean 633 wrote:

What the most stronger T3 ? T3 from armour or synthetic T3, or is the same ?

They are both the same, pound for pound, mcg for mcg.

[DrMariano2]

@Jean 633 wrote:

What the most stronger T3 ? T3 from armour or synthetic T3, or is the same ?

They are the same.

[Mebigusmall]

when one supplements only T4 and does not use armour, are they getting T1 and T2? At what point in the process is T1 and T2 created? I seem to remember you saying T1 is used in the brain, is that correct.

[hardasnails1973]

One can take substitute 1/2 grain of armour for 50 mcgs of t-4 or vice versa and get the benefits. Since adding more t-4 I am so much more emotional and mentally stable then before.

[Mebigusmall]

So your saying hald armour and half T4? and does this yeild different results than using
T4 and T3 that is not dessicated?

[Author]

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